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TB 1000 injection

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TB 1000 injection is a peptide which is applied by subcutaneous injection to promote angiogenesis, wound healing muscle growth.

Description

TB 1000 injection is a peptide which is applied by subcutaneous injection to promote angiogenesis, wound healing muscle growth. It has demonstrated cardio protective and immune-potentiating effects, as a potent anti-aging, and has anti-scarring properties TB-1000 is a synthetic version of the naturally occurring peptide present in virtually all human and animal cells first located in the Thymus gland in 1981. The potency of Thyrosin Beta 4 present in TB-1000 is approximately 99%, the highest of its kind.

How does TB-1000 Work?

In angiogenesis and in wound healing, TB-1000 acts by accelerating the migration of blood vessel cells and skin cells and increasing the production of extracellular matrix-degrading enzymes. It has also been shown to stimulate epidermal stem cell differentiator. When applied by subcutaneous injection, it assists in the generation of new blood vessels and aids wound healing by promoting skin cell regeneration.

How does TB-1000 Work Scientifically?
One of TB-1000 4’s key mechanisms of action is its ability to regulate the cell-building protein, actin, a vital component of cell structure and movement. Of the thousands of proteins present in cells, actin represents up to 10% of the total proteins which therefore plays a major role in the genetic makeup of the cell.
This potent peptide is a member of the ubiquitous family of 16 related molecules with
a high conservation of sequence and localization in most tissues and circulating cells
in the body.not only binds to actin, but also blocks actin polymerization and
is the actin-sequestering molecule in Eukaryota cells.
TB-1000 was identified as a gene that was up-regulated four to six fold during early
blood vessel formation and found to promote the growth of new blood cells from the
existing vessels.
This peptide is present in wound fluid and when administered subcutaneously, it
promotes wound healing, muscle building, decreases recovery time of muscles fibers
and their cells.
An additional key factor of is that it promotes cell migration through a specific
interaction with actin in the cell cytoskeletal. It has been demonstrated that a central
small amino acid long actin binding domain has both blood cell reproduction and
wound healing characteristics. These characteristics are uncovered by accelerating the
migration of endothelial cells and keratinocytes. It also increases the production of
extracellular matrix-degrading enzymes.

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